Skip navigation

Lori Reyna

​​​​Photo of Kevin PangleLecturer II
Biology
Biosciences 3109
989-774-2648
​​
Education
  • Ph.D., University of Miami, 2002
  • B.S., Central Michigan University, 1998
Teaching Areas
  • ​Introductory Biology
  • Biotechnology
  • Molecular & Cell Biology


Areas of Interest
  • Molecular Pharmacology
  • Apoptosis
  • Protein & Nucleic Acid-Based Therapeutics


Patents
  • Dendritic Polymer Conjugates Having a Multi-Chromophoric Surface (2007; 60/926,626
  • Delivery of Biologically Active Materials Using Core-Shell Tecto (Dendritic Polymers) (2007); 60/779,267
  • Dendritic Polymers with Enhanced Amplification and Interior Functionality (2005); PCT/US2005/047635
  • Process for Preparing Bio-Complexes Using Dendritic Polymers (2004); 60/588,824


​​Selected Publications
  • Tomalia, D.A., Reyna, L.A., and Svenson, S. (2007) Dendrimers as Multi-Purpose Nanodevices for Oncology Drug Delivery and Diagnostic Imaging. Biochem. Soc. Trans. 35(Pt 1), 61-7. 
  • Chauhan, A.S., Svenson, S., Reyna, L.A., and Tomalia, D.A. (2007) Solubility Enhancement of Poorly Water Soluble Molecules using Dendrimers. Material Matters. Advanced Applications of Engineered Nanomaterials. 2(1), 24-26. 
  • Kubasiak, L.A. and Tomalia, D.A. Cationic Dendrimers as Gene Transfection Vectors:Dendri-Poly(amidoamines) and Dendri-Poly(propylenimines). Polymeric Gene Delivery: Principles and Applications. M.M. Amiji, ed. CRC Press, July 2004. 
  • Kubasiak, L.A., Frazier, D., Dougherty, C. J., Li, H., Bishopric, N.H., and Webster, K.A. (2004) Mitochondrial Signals Initiate the Activation of c-Jun N-terminal Kinase (JNK) by Hypoxia-Reoxygenation. FASEB Journal. 18(10), 1060-1070. 
  • Kubasiak, L.A., Hernandez, O.M., Bishopric, N.H., and Webster, K.A. (2002) Hypoxia and Acidosis Activate Cardiac Myocyte Death through the Bcl-2 Family Protein BNIP3. Proc. Natl. Acad. Sci., USA. 99(20), 12825-30. 
  • Dougherty, C.J., Kubasiak, L.A., Prentice, H., Andreka, P., Bishopric, N.H., and Webster, K.A. (2002) Activation of c-Jun N-terminal Kinase Promotes Survival of Cardiac Myocytes After Oxidative Stress. Biochem. J. 362(Pt3), 561-71.